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Functionally, persistent cardiac mitochondrial dysfunction is consistent with restricted ATP production, that could impair contractility and increase susceptibility to arrhythmogenesisfindings consistent with chest pain, palpitations, and POTS observed clinically (56)
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Stimulate energy and metabolism by replenishing T3 and T4
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In perfused pancreas models, it has been observed that GLP-1 significantly inhibits glucagon secretion from pancreatic -cells across various glucose levels
