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GLP-1 is produced by l-cells primarily in the distal parts of the intestine, but it can also activate a Gs-coupled GLP-1 receptor (GLP-1R) resulting in proliferation of human coronary artery endothelial cells as well as -cells in pancreas (Egholm et al., 2016)

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Biotinidase Deficiency Profound biotinidase deficiency is an autosomal recessive disorder that is characterized by mutations in the BTD gene that result in enzyme activity that is less than 10% of the normal

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HGF protects endothelial cells from hypoxic death by the AKT pathway and p38 MAPK , as well as through the enhancement of Bcl-2 expression (Yamamoto et al., 2001