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Several basic molecular and cellular mechanisms are shared among brain diseases, such as neuroinflammation, oxidative stress, excitotoxicity, neurodegeneration, disorders of neurogenesis, dysfunction of neurotrophic factor systems, which are most prominent in specific brain structures associated with different brain diseases [104, 105]

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These pathways are examined using cellular, molecular, and electrophysiological assays to map receptor-dependent signaling under controlled laboratory conditions

For the peptide's mechanism and research bibliography, visit the GHK-Cu peptide page

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Exp Neurol 1996