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Through co-immunoprecipitation (CO-IP) experiments, we discovered that FOXO4-DRI prevents the binding of FOXO4 to P53, facilitating the phosphorylated P53 nuclear exclusion, which subsequently trigger BAX and cleaved caspase-3, leading to the apoptosis of senescent cells

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(Mechanisms) Several complementary mechanisms have been proposed from cell and animal studies: Angiogenesis & Microcirculation : In damaged tissue, BPC-157 appears to support pro-healing neovascularisation , restoring local perfusion

Langan, R., & Goodbred, A

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