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10.1186/s40168-021-01107-9 304 ZhaoZ.YanJ.HuangL.YangX

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Funding This work was partially supported by the Puerto Rico Science, Technology and Research Trust award, and the following grants from the National Institutes of Health: NIGMS/MBRS Grant GM08224, NIMHD RCMI-Infectious and Global Diseases Center Grant G12MD007600, and NIMHD CCRHD-Tropical Emerging Infectious Diseases Research Center Grant U54MD007600

Mechanistically, GHK-Cu preserved mitochondrial function by increasing mitochondrial membrane potential, alleviating age-related mitochondrial network fragmentation, shifting mitochondrial dynamics toward fusion via regulating drp-1 and fzo-1 expression, and promoting ATP biosynthesis

The ghrelin receptor pathway Ipamorelin mimics ghrelin, the "hunger hormone" produced primarily in the stomach