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Research indicates that excessively hyperplastic and dysfunctional EAT exerts adverse cardiac effects primarily through two pathways: first, by serving as a source of pro-inflammatory mediators, and second, through direct cardiac mechanical effects, ultimately promoting cardiovascular disease (82)

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In the United States, 10.5% of the population has diabetes, with about 9095% of those having T2D [2]

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As mentioned earlier, decreased skeletal muscle mass may trigger the body to unload our bone density

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