DNase-I treatment showed more single colonies uniformly distributed on the surface, whereas CLSM images revealed amylase and Proteinase treatment showed a mixture of small and big clusters in biofilm architecture (Figure 3C)
Induced mitochondrial dysfunction caused by oral anticoagulation, such as warfarin interacting with coenzyme Q10, is questioned [364,365] (Figure 3)
The pattern of liver injury induced by methimazole is predominantly as cholestatic type,21,22,23 where those induced by PTU is of hepatocellular type in most cases.20,24,25 Pathologic findings of PTU-induced hepatic injury in human subjects revealed that this drug caused parenchymal necrosis with hemorrhage, collapse of lobular architecture, and preportal mixed inflammatory infiltrate.26 Hepatic abnormalities associated with methimazole are typical of a cholestatic process.22 Biopsy specimens from patients with methimazole-induced hepatic injury showed oedema and inflammation of portal tract with intracanalicular cholestasis and moderate microvascular steatosis.27,28 Fewer studies on the mechanisms and the pathogenesis of liver damage induced by these drugs are available
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The global prevalence of glucose-6-phosphate dehydrogenase deficiency: a systematic review and meta-analysis
