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Collectively, our in vivo and in vitro findings are consistent with TMAO aggravating kidney fibrosis due to ERK1/2 hyperactivation synergistically, probably with activation of the TGF-1-mediated SMAD3 pathway causing a secondary hit in our disease model

3 NADPH usage for antioxidative purposes While cytotoxic reactive radicals produced by myeloid cells are essential for limiting bacterial replication, they can also be harmful to the host (34, 35)

doi: 10.1111/obr.12626 420 SuzumuraEABersch-FerreiraACTorreglosaCRda SilvaJTCoqueiroAYKuntzMGet al