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To further determine whether depleting -catenin could rescue the differentiation defect caused by Ssu72 loss, we performed a double knockdown of -catenin and Ssu72 in 3T3-L1 cells, followed by induction of adipocyte differentiation

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Ultimately, this pattern of compulsive seeking and consumptiondriven by neurobiological mechanismssignificantly contributes to the perpetuation of obesity [25], posing a challenge for interventions that do not effectively modulate the glycemic and insulinemic responses underlying these mechanisms

Mechanisms of GLP-1 Receptor Agonist-Induced Weight Loss