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The Plant Journal 43, 900914

First elinical experiences with superoxide dismutase in kidney transplantation

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However, it may be because extracellular matrix (ECM) accumulation and apoptosis work together in several different pathogenic mechanisms to cause the injury and progression of kidney disease ( , thus inducing oxidative stress with an attendant increase in apoptosis-related protein expression and subsequent kidney cell apoptosis ( In vitro studies demonstrate that arsenic (As) export relies on the interaction between glutathione (GSH) and As, forming a complex