Previously, we showed that TG2 mediates crystallin, caspase 3, and collagen crosslinking in response to oxidative stress, hypoxic stress, and DNA damage, which leads to cataract formation, apoptotic resistance, and fibrotic development, respectively 4,5,6
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Its primary mechanism of action involves the activation of telomerase, which helps maintain the length and integrity of telomeres
Annual Review of Biophysics and Bioengineering, 7, 435468
These findings are further supported by Anitha et al., who observed the efficacy of C
All infusions are customized and administered under physician supervision