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PGC-1alpha expression decreases in the Alzheimer disease brain as a function of dementia

Pharmacovigilance reports do show signals that have not been fully explained, and the question is not entirely settled

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These findings suggest that GABA can mitigate abnormal metabolism in the striatum via pathways such as arginine biosynthesis, aminoacyl-tRNA biosynthesis, GABAergic synapse, D-amino acid metabolism, central carbon metabolism in cancer, and protein digestion and absorption (Figure 3E)