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cGAS/STING pathway in microglial diseases In the context of neurodegeneration, amplification of microglial cGAS-STING signaling, along with IFN production and cGAS-driven senescence, has been implicated as a disease-promoting mechanism in female mice ( R47H variant, indicating both associated with increased risk and earlier onset of Alzheimer's disease and their potential role as upstream modulators of AD pathogenesis (Yu et al., 2020)

K.ScheunemannL.EisenbergT.MertelS.BhukelA.KoemansT

Stores sit mainly in the liver and last years, so deficiency develops slowly and quietly, which is precisely why it gets missed

The Phase 2 trial data was striking

R.Hojjati KermaniM

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