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Recent work suggested that the reduction in both food intake and body weight observed in response to combined treatment with GLP-1RAs and PYY 3-36 results in synergistically-enhanced activation of discrete hypothalamic and brainstem circuits that regulate appetite, including the arcuate nucleus (ARC), the paraventricular nuclei of the hypothalamus (PVNs), the central nucleus of the amygdala (CeA), and the hindbrain (AP/NTS) 10,12,21,44

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Little is known about whether the global shortages have prevented patients from accessing their prescribed anti-diabetic (ADM) or anti-obesity (AOM) medications or switching to other GLP-1 RA medications

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