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On one hand, OSCC tissues universally display a ferroptosis-resistant phenotype typified by up-regulated GPX4 and high SLC7A11 expression ( inhibitors and GPX4 inhibitorshave both been validated in OSCC models

Every decade, levels drop by another 10 to 15%

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Ferroportin-mediated ferroptosis involved in new-onset atrial fibrillation with LPS-induced endotoxemia

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