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Strengthening this translational bridge between immunometabolism and infection biology will be crucial for developing personalized, durable solutions to these intertwined global health challenges

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Thus, based on the clear and direct role of Y1-R in promoting glucose uptake in muscle, the lack of any response to GLP-1R agonism, and the glucose-reducing effects of GEP44 observed in our previous studies 16 , we predicted that GEP44 would also promote glucose uptake via interactions with the Y1-R

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Reduces Inflammation Inflammation is a natural response to injury or infection