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This spatial and transcriptional heterogeneity suggests that localized ferroptotic events may drive tissue damage by interfering with cartilage repair and intercellular communication

The interaction of gliadin and its immunomodulatory fragments with chemokine CXC receptor 3 on IECs induces zonulin release and downstream disassembly of TJs which has been reported previously among coeliac patients (Lammers et al

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Creating the amylin story