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Conclusion We performed lipidomic profiling of oxylipins originating from different precursors including epoxides, diols and hydroxy fatty acids

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& Chau, Y

GIPR antagonism and GLP-1R agonism both modulate DVC gene programmes implicated in synaptic plasticity We next performed cell-type prioritization analysis using Augur 42 to determine which types of neurons were most affected by individual drug treatment in the DVC (Fig