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10.3109/00016489.2011.577448 Acta Otolaryngol

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The mechanisms that drive enteroendocrine release of these gut peptides into the blood stream are multifactorial and include responses to direct nutrient stimulation, intracellular metabolism, or neuroendocrine mechanisms [see Psichas, et al

This extensive binding dramatically reduces the uptake of native IGF-1, limiting its interaction with target tissue receptors

PPAR is a major transcriptional regulator of fatty acid oxidation and extended PPAR activation causes HCC in rodent mice by mechanisms that involve perturbation of the cell cycle and production of ROS [reviewed by Michalik et al