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In addition, agonist binding increases NO-antiaggregating effects, cGMP/PKG/VASP pathway activation, reduces of PI3-K/Akt and MAPK/ERK-2 pathways activation, and decreases arachidonic acid (AA)-induced oxidative stress [93, 94] (Fig

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Relative to rAmy bound to CTR, Cagri is likely to require extra energy to weaken the salt bridge to attain a CT-like state, thus providing a structural rationale for the smaller population of Cagri-bound particles sampling a CT-like conformation

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