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In a range of AD animal models, treatment with GLP-1, GIP, or dual-GLP-1/GIP receptor agonists effectively protected cognition, synaptic trans-mission, long-term potentiation (LTP), and prevented the loss of synapses and neurons

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Shorter dendrites and reduced synapse pruning are observed in different ASD models, such as prenatal exposure to selective serotonin transporter inhibitors (SSRIs) and maternal inflammation

Simultaneously, these drugs suppress glucagon secretion, which prevents the liver from releasing excess glucose into the bloodstream

High concentrations of resveratrol may inhibit mitochondrial complex I, while synthetic activators like SRT2104 require high doses due to poor liver targeting, increasing oxidative stress risk in extrahepatic tissues