Ferroptosis, through its associated oxidative stress and iron dysregulation, can reciprocally modulate autophagy, forming a dynamic bidirectional network crucial for cell fate decisions in the tumor microenvironment
A construct was prepared by joining the amplified gene fragments of CFP, YliB and YFP in pGEM-T easy vector
Once carnitine metabolism is disrupted, it is not only a reflection of impaired renal function, but also may promote the occurrence and progression of DKD through mitochondrial energy dysfunction, oxidative stress and inflammatory responses
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