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While the inflammation-reducing and fibrosis-attenuating effects of glucagon agonism are less well-characterized, the reductions in hepatic inflammation parameters and fibrosis observed in preclinical models likely reflect secondary effects consequent to improved steatosis, rather than direct anti-inflammatory actions [21]

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These medications mimic a natural hormone in the body known as GLP-1

However, TGF- signaling can suppress AhR expression via methyl-CpG-binding domain 2 (MBD2) -mediated promoter methylation, potentially limiting the antifibrotic efficacy of Kyn [117]

13, 674135 10.3389/fnagi.2021.674135 [DOI] [PMC free article] [PubMed] [Google Scholar] 151.Iciek M., Chwatko G., Lorenc-Koci E., Bald E

myl7:EGFP) , which carried pathogenic LMNA variants that had been previously isolated and reported to cause EDMD 29