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Studies have shown that ROS can oxidize the cysteine residues in the amino acid transporter LAT1, which can affect its function and thus influence the entry of amino acids into cells [201, 202]

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In cases where ligands generated a large signal during the association phase (at least 300% greater than the negative control) but could not be reliably fit, binding classification was assigned based on the observed magnitude of the shift

Therefore, riboflavin deficiency will lead to the downregulation of GSH and oxidative stress, which is the predominant cause for reduced osteogenic capacities of BMSCs and enhanced osteoclastic potentials of osteoclast precursors [54, 55], finally leading to net bone loss

The benefit persisted at the 12-month follow-up