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Although the mechanisms underlying these effects remain largely unknown, they are assumed to at least in part rely on the ability of PPAR to decrease ectopic lipid deposition by stimulating adipocyte differentiation and fatty acid uptake 10,11,23

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has received honoraria for trial leadership from Amgen, Boehringer Ingelheim, Lilly USA, Metsera, NewAmsterdam, Novartis, Novo Nordisk and Pfizer, and honoraria for consultancy from Alveus, Amgen, Boehringer Ingelheim, F

Varanasi et al , 2011), suggesting that GLP1R expressed in cells, outside of beta cells, may also have a role in improving glycemic levels even in people who functionally lack beta cells and would ultimately mean lower need for insulin in T1DM patients