[DOI] [PMC free article] [PubMed] [Google Scholar] Ichijo H, Nshida E, Irie K, ten Dijke P, Saitoh M, Moriguchi T, Takagi M, Matsumoto K, Miyazono K and Gotoh Y
Moreover, this evidence suggests that the ability to exploit both glycolysis and OXPHOS not only supports tumor growth under suboptimal conditions but also confers resistance to treatments designed to target one metabolic pathway
Reduced thiols Results are detailed in the Table 6
Stojkovska I, Krainc D, Mazzulli JR
Then, the massive accumulation of ROS set up a sequence of events that led to cell death in the brain
In addition, GLP-1 activation in astrocytes by GLP-1R altered cellular glucose metabolism, revealing a novel mechanism by which GLP-1R improves cognitive function in patients with AD (Zheng et al., 2021)