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High MG levels can change the membrane potential of mitochondria, lower ATP production, and trigger cell death pathways related to oxidative harm (76)
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They are glucagon-like peptide agonists, or GLP-1s, though technically I should be calling them GLP-1As
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Hornigold (Cardiovascular, Renal, and Metabolism Research, MedImmune Ltd., Cambridge, UK) who contributed to the in vitro experiments described in the ESM
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