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Gliadin-dependent neuromuscular and epithelial secretory responses in gluten-sensitive HLA-DQ8 transgenic mice
They have become essential components for optogenetic applications, where they can be fused to different output domains to provide light control of various cellular functions 1
Taken together, our bioinformatic approach identified a network of genes involved in HIF biology that are accessible for transcriptional expression in the human kidney, supporting an environment inherently responsive to hypoxic conditions
On the contrary, high numbers of invading bacteria exert an overactivation of Toll-like receptors (TLRs), leading to the overexpression of inflammatory cytokines, with consequent epithelial damage and chronic inflammation [10]
This more then piqued my interest
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