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In vivo, exenatide treatment of orthotopic CT26 tumors showed no change in tumor weight

MC3R activation has been investigated in the context of energy balance feedback, natriuretic signaling, and modulation of inflammatory mediator release (Butler et al., Endocrinology, 2000

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Taken together, these data suggest that inhibiting HGF does not alter the number of functional synapses in vehicle treated neurons but attenuates the effects of HGF and Dihexa on synaptogenesis by decreasing the number of postsynaptic spines

94 GLP-1 RA has been shown to exert anti-inflammatory effects in the CNS

However, this combination did not appear to improve survival for metastatic or advanced PDAC patients [100]