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Normally expressed as an inactive monomer in the cytoplasm, BNIP3 forms a stable homodimer via its C-terminal TM domain under stress, integrating into the OMM [12]

In this context, caution is warranted in extrapolating the physiological correlates of early treatment response as indicators of the emergence of categorical treatment resistance at later stages of schizophrenia

Meanwhile, various CCKR-targeted drugs have been developed, yet most of them were ultimately removed from the market due to questionable efficacy, lack of target specificity, or severe adverse effects 28,29