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Dempsey's published article about GLP-1 treatments in MCAS is here

Each individual treatment plan is personalized based on a persons body, health history and overall health and wellness goals

At the same time these astrocytes reduce expression of several key homoeostatic proteins such as glutamate transporters, glutamine synthetase, K ir 4.1 inward rectifying channels involved in K + buffering, and connexion 43 responsible for syncytial coupling

When you sleep well, your body can make more GLP-1, which helps with hunger control and metabolism

Continuing treatment at a maintenance dose may help support steady appetite signals and weight stability, and studies show that people who stay on the medication are often better able to maintain their weight than those who stop treatment completely

We know how isolating it can be