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Furthermore, GLP-1 RAs play a pivotal role in the alleviation of oxidative stress, the maintenance of mitochondrial homeostasis, and the inhibition of cell apoptosis and pyroptosis in models of ischemic cardiomyopathy [16]

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The data conclusively demonstrated that both ME/CFS and Long COVID are characterized by severe, elevated oxidative stress and profound mitochondrial damage, forcing immune cells to consume excess host energy

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