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Diabetologia, 60, 15771585

Acknowledgements This research is supported by the Research Grant of the University of Tabriz (number S/4829-14020128)

Consistently, AMPK activator AICAR was attenuated, while PARP-1 over-activation aggravated cholesterol-induced toxicity in INS-1 cells, indicating that in islet cells, PARP-1 also regulates cholesterol efflux through AMPK activation and ABCA1 protein, therefore affecting the GLP-1 protection against cholesterol-induced toxicity

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