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This metabolic bottleneck places cysteine availability at the center of cellular redox adaptation and makes it a critical determinant of GSH homeostasis under stress conditions

Our APAP model, combined with our GSH model, gives predictions about humans

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Therefore, drugs that stimulate GLP-1 receptors and inhibit GLP-1 breakdown, exenatide, and sitagliptin, respectively, reduce mices self-administration of nicotine