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doi:10.3390/ijerph18157911 Note: Originally published in October 2017, this article has been revised and updated for accuracy and thoroughness.
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These findings collectively suggest that GLP-1 agonists, such as semaglutide, may represent a novel therapeutic strategy for RV dysfunction by targeting mitochondrial pathology and restoring critical signalling pathways like GSK3-Drp1
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Furthermore, significantly elevated branch-chain fatty acids (BCFA), which are metabolic products of protein breakdown have been hypothesized to induce epithelium inflammation ( These microbial alterations coincided with PTSD symptomatology, BBB dysfunction and hippocampal neuroinflammation, which was confirmed by increased proinflammatory cytokine levels IL-1 and IL-6 (Tanelian et al., 2023)
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