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Following the emergence of MDM2 as a p53 inhibitor, pharmacological disruption of the interaction between these two proteins has been explored to reactivate p53 in human tumors that express wild-type TP53

However, the secondary follicle number was significantly higher in the L-carnitine-supplemented group (G2) compared to G1 and G3

Limitations of the Existing Evidence Route mismatch

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Infectious agents Extensive efforts have been made, with limited success, to establish an infectious agent as the cause of IC

There was no verification process to ensure I was a researcher