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These findings reveal that adipocyte circadian disruption impairs metabolic health through mitochondrial complex I dysfunction, establishing clock control of complex I as a key regulator of metabolic homeostasis

Detailed examination of a subset of these patients indicates that these metabolic abnormalities are at least partly due to TCAC and ETC dysfunction

Research points to GLP-1 as having a role in entraining the circadian rhythm of insulin release in the pancreas