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Continuous activation of TNF- leads to synaptic dysfunction, neuronal loss, and worsening of amyloid and tau pathology

Given that gut microbiota imbalances may contribute to AD progression by promoting amyloid-beta accumulation, triggering inflammation, increasing oxidative damage, and impairing insulin signaling 45,82 , these findings highlight the possibility that modulation of gut-brain axis may be a key mechanism underlying the cognitive benefits of dual-action therapies in DM2-AD comorbidity

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It interacts with the brains hunger centers to promote feelings of fullness, effectively reducing the desire to overeat

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