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In retinal endothelial cells, activation of the GLP-1 receptor increases PI3K/Akt signaling, which activates endothelial nitric oxide synthase (eNOS), leading to NO production

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Jones said his future research will focus on evaluating GLP-1 therapies in other high-risk gastrointestinal cancers and on designing long-term prospective studies to determine whether these medications could one day play a role in primary cancer prevention

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