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The inheritance of GDH-HI is mainly autosomal dominant or secondary to de novo mutations, of which autosomal dominant inheritance accounts for ~ 20% while de novo mutations account for ~ 80% of all cases [11]
To view a copy of this licence, visit About this article Cite this article Kabir, A., B., M., Manohar, A
Violi, F
Without GSH depletion, viability of mock- and GSTP1 -transfected cells was comparable for all treatments
With major vascular occlusions, there was, at the periphery, the leading role of the rapid heart disturbances, and thereby, heart, lung, liver, kidney and gastrointestinal lesions, inferior and superior vena caval congestion, azygos vein failure, portal and caval hypertension and aortal hypotension occur [14,15,16,17,18,19,20]
Moreover, efpeglenatide therapy reduced the risk of CV disease by about 27% compared to the placebo in the Effect of Efpeglenatide on Cardiovascular Outcomes (AMPLITUDE-O) study [51]
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