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When an oxygen molecule enters the active site, deoxy tyrosinase converts back to the oxidized tyrosinase state, completing the cycle

2015;29(1):no.385.2

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Timely self-elimination of damaged mitochondria from cells through mitophagy is an important quality control mechanism for cell homeostasis and survival during injury and repair [42]

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Published research suggests this interaction may be bidirectional, with BPC-157 appearing to promote or counteract NO system effects depending on the experimental context (Sikiric et al., 2014)