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Given that YAP is a transcriptional co-activator known to translocate into the nucleus in response to ECM stiffening in various cell types, including melanoma cells (Miskolczi et al, 2018), and that YAP may serve as a potential MITF partner in uveal melanoma cells (Barbosa et al, 2023), we wondered whether YAP could be associated with the observed ECM-dependent MC phenotypes

How This Works Semaglutide is a GLP-1 receptor agonist with 94% sequence homology to native human GLP-1 [4]

MSSM-002 reduced GATA-3 mRNA and protein expression and the binding to the IL-4 gene promoter in D10 cells [94]