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observed that semaglutide treatment increased UCP1 labeling, promoted thermogenic gene expression necessary for maintaining the browning phenotype: beta-3 adrenergic receptor (+520%), PR domain-containing 16 (+90%), and UCP1 (+110%) while reducing inflammatory markers [11]
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In contrast, the triagonist induced a significant decrease in ovarian and uterus weight vs
The observable consequence is the most potent weight- and glucose-lowering profile of any molecule approved before retatrutide: SURMOUNT-1 reported roughly 22.5% weight reduction at the 15 mg dose over 72 weeks 13, and SURMOUNT-5 showed tirzepatide superior to semaglutide 2.4 mg head-to-head at 72 weeks (20.2% vs 13.7%) 18
