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[1] Together with toxic epidermal necrolysis (TEN) and StevensJohnson/toxic epidermal necrolysis (SJS/TEN) overlap, they are considered febrile mucocutaneous drug reactions and probably part of the same spectrum of disease, with SJS being less severe

GSH could also be converted to oxidized GSH (GSSG) via various glutathione peroxidases (GPX) in this process (Figure 3)

Copper homeostasis and cuproptosis represent emerging targets for therapeutic intervention in inflammatory diseases

demonstrated that IL-6 could contribute to the regulation of VEGF and angiogenesis in GC

Here both supplements are generally discouraged