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PEM is also thought to be linked to insufficient ATP levels due to a lack of available adenosine, which delays the rapid replenishment of ATP stores post-exercise and may contribute to prolonged recovery times observed in these patients following physical exertion (Lengert and Drossel, 2015)

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244 Polymorphisms in these genes may lead to alterations in collagen synthesis, cross-linking, and fibril formation, resulting in weaker collagen fibers and an increased risk of ligament injuries

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