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meningitidis rapidly colonized capillaries and arterioles before the activation of an efficient inflammatory response

No direct causal link has been established between GLP-1R use and increased aspiration risk

Intracellular glutathione also followed the trend of GCLC, exhibiting an NFT-dependent increase that was reversed by anti Nrf2-siRNA to levels comparable to those observed in untreated HepG2 cells (Fig

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