While Semaglutide was originally developed for type 2 diabetes, its FDA-approved specifically for weight loss in non-diabetic patients
And while metformin has been the first-line therapy in treating type 2 diabetes, the FDA announced in 2019 that Rybelsus (semaglutide), a GLP-1, can also be offered as a first-line treatment
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[1] Whilst semaglutide alone rarely causes hypoglycaemia due to its glucose-dependent mechanism, a rapid bolus effect combined with other glucose-lowering medications could theoretically increase this risk
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The Proposed GLP-1 Agonist POP Toxin Redistribution Axis In Short Bioaccumulation of POP chemicals into adipose fat from normal to high exposures GLP-1 agonist drug induces rapid adipose fat burning Rapid lipolysis of adipose fat releases stored POPs into the bloodstream in high amounts, and over prolonged periods of time POPs recirculate bound to lipoproteins and to a lesser extent, albumin With the substantial loss of adipose fat, POP toxins are not adequately bio-transformed, but instead redistributed to other lipid-rich organs, cells and tissues Abstract Research studies on lipophilic, persistent organic pollutants (POPs) such as Dioxin, PCB, PBDEs and HCB have demonstrated that these types of toxins are predominantly stored in adipose fat
