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Basic Neurochemistry NCBI Bookshelf

Q6: What are the main side effects of GHK-Cu

Frontiers in Neuroscience 8 (2014)

ACTL6A is found to be critical for regulating the glutathione (GSH) metabolism pathway because it upregulates -glutamyl-cysteine ligase catalytic subunit (GCLC) expression, thereby reducing reactive oxygen species (ROS) levels and inhibiting ferroptosis, a regulated form of cell death driven by the accumulation of lipid-based ROS

doi: 10.1096/fasebj.8.15.8001743

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