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Point mutations or deletions of the -globin gene cluster, which is located on the human chromosome 11 p15.3 locus, induce -thal pathogenicity by reducing or eliminating -globin synthesis (Taher et al., 2018)

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The effects of Exendin-4 were abolished in the presence of Exendin (939), but remained intact when insulin secretion was abrogated by diazoxide (Fig

2,9 DXA, as a molecular-level assessment, provides estimates of LST (i.e., total mass minus fat mass (FM) and bone mineral mass) or FFM (total mass minus FM)

Multimodal treatment of an extremely rare desmoplastic small round cell tumor primary to the brachial plexusA case report and review of literature

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