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Our results demonstrate the re-purposing of an endogenous housekeeping gene to form a family of effectors with modified functions

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For an analysis of the risk associated with the deletion polymorphism for GSTT1 , it was found that the null genotype (p = 0.0004) is related to an increased predisposition for T2DM (Table 2), conferring a 3.2-fold increased risk of developing the disease relative to the present genotype

and acute and chronic lymphocytic leukemias

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